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Original Question

what is the purpose of this article? Diabetes in children and adolescents is now acknowledged to be a complex disorder with heterogeneity in its etiology, pathogenesis, clinical presentation, and outcomes. The majority of pediatric diabetes is classified into one of two broad categories according to physiologic framework established by the American Diabetes Association (ADA) in 1997:1 type 1 diabetes (T1D), an absolute deficiency of insulin usually due to autoimmune destruction of the beta cells, and type 2 diabetes (T2D), a combination of insulin resistance and relative insulin deficiency.2,3 Until recently, diabetes diagnosed in children and adolescents was almost entirely considered to be T1D, but T2D has recently emerged among youth with obesity and in high risk ethnic populations.4,5 Although there are other forms of diabetes that affect youth, including genetic defects of beta-cell function, genetic defects of insulin action, and a variety of secondary forms of diabetes3,6, this chapter focuses on the most common forms of pediatric diabetes: T1D and T2D in youth.

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