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summarize and simplify Currently, therapeutic intervention for acute heart failure (HF) is initiated only after the appearance of clinical signs that characterize the alveolar stage of pulmonary edema (APE). Advanced stages of APE cause considerable distress to patients and are difficult to treat because of a vicious cycle in which deteriorating arterial oxygen saturation due to pulmonary congestion leads to failing myocardial function which, in turn, leads to further decrease in oxygen saturation.[1] Preventive treatment (PT) of APE might interrupt its development by aborting the vicious cycle that progressively leads to HF.[2] The only possible solution for early diagnosis (i.e., before the appearance of clinical symptoms) and PT of APE would be continuous monitoring of every patient at risk of developing acute HF. The existing methods of detecting APE during the preclinical stage are invasive, not reliable enough and cannot be used for predicting APE.[3]-[8] The measurement of pulmonary capillary wedge pressure is invasive and certainly can not be used for the purpose of monitoring all cardiac patients at risk of APE, especially those who are outpatients. Similarly, X-rays can not be used for prolonged monitoring due to the danger of frequent exposure to radiation.

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